KELP reduces Inflammation in UC and Crohn's

Brown algae (Fucus and Ascophyllum nodosum) has proven anti-inflammatory properties. Inflammatory diseases such as PsoriasisCrohn's disease and Ulcerative colitis are characterized by an excess of white blood cells. It has been demonstrated that the spread of leukocytes can be slowed when taking fucus drugs.

Fucoidan's action eliminates the root cause of inflammation - modified cells in the stomach and intestines.  As a result, one is freed from the inflammation and accompanying erosive and ulcerative changes, as well as the risk of cancer.

 

Fucoidan acts as inhibitors of inflammatory mediators, almost like a new generation of drugs for rheumatoid arthritis, inflammatory bowel disease (IBD) and psoriasis.

A large body of RESEARCH across a number of disease areas shows that the more acute infections you get, the less your risk of cancer and autoimmune diseases such as Crohn’s disease and ulcerative colitis. Acute infection stimulates the body’s innate immune system (macrophage function).

There is growing evidence that

a defect or deficiency of the innate immune system may be an important underlying cause of both Cancer and autoimmune diseases such as

Ulcerative Colitis and Crohn’s disease.

Macrophages are important cells of the body’s innate immune system. One of their primary roles is to engulf (through phagocytosis) dying or dead cells, in order to clear/recycle these dead and dying cells. The cells of the tissues in our body have a natural life cycle, measured in days, weeks or months depending on the tissue. When a cell reaches the end of its natural life cycle, it signals to macrophages that it is dying.

Apoptosis, the process of self-destruction of transformed cells, is inviting macrophages to engulf and recycle the cell. When this happens, the macrophage engulfs the cell and sends out anti-inflammatory and ‘tolerance’ signals so that the adaptive immune system (the other major arm of the immune system) doesn’t react against the antigens of the dying cell.

However, if there is a defect or deficiency of macrophage function (innate immune system), especially if there is an environmental trigger such as stress or chronic infection that overwhelms macrophage function, macrophages don’t clear dying or dead cells efficiently.

As a result, not all dying or dead cells are cleared by macrophages and, when they aren’t cleared, these dead cells necrose releasing their internal antigens to which the adaptive immune system reacts, resulting in antibodies against these self-antigens and autoimmune disease (such as UC and Crohn’s disease).

In other words, while symptoms of autoimmune disease are generally understood to be a result of an over-reactive adaptive immune system response, the underlying trigger/cause may be a defect or deficiency in the innate immune system (macrophage function). 

Source*


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